dc.contributor.author |
Barrett, Carolyn |
en |
dc.contributor.author |
Ramchandra, R |
en |
dc.contributor.author |
Guild, Sarah-Jane |
en |
dc.contributor.author |
Lala, A |
en |
dc.contributor.author |
Budgett, DM |
en |
dc.contributor.author |
Malpas, Simon |
en |
dc.date.accessioned |
2011-09-04T21:48:57Z |
en |
dc.date.issued |
2003 |
en |
dc.identifier.citation |
Circ Res 92(12):1330-1336 27 Jun 2003 |
en |
dc.identifier.issn |
0009-7330 |
en |
dc.identifier.uri |
http://hdl.handle.net/2292/7619 |
en |
dc.description.abstract |
Increasing evidence suggests elevated sympathetic outflow may be important in the genesis of hypertension. It is thought that peripheral angiotensin II, in addition to its pressor actions, may act centrally to increase sympathetic nerve activity (SNA |
en |
dc.description.uri |
http://circres.ahajournals.org/cgi/content/full/92/12/1330 |
en |
dc.language |
EN |
en |
dc.relation.ispartofseries |
Circulation Research |
en |
dc.rights |
Items in ResearchSpace are protected by copyright, with all rights reserved, unless otherwise indicated. Previously published items are made available in accordance with the copyright policy of the publisher. Details obtained from http://www.sherpa.ac.uk/romeo/issn/0009-7330/ |
en |
dc.rights.uri |
https://researchspace.auckland.ac.nz/docs/uoa-docs/rights.htm |
en |
dc.subject |
rabbits |
en |
dc.subject |
telemetry |
en |
dc.subject |
angiotensin II |
en |
dc.subject |
baroreflex |
en |
dc.subject |
sympathetic nerve activity |
en |
dc.subject |
SPONTANEOUSLY HYPERTENSIVE RATS |
en |
dc.subject |
ROSTRAL VENTROLATERAL MEDULLA |
en |
dc.subject |
ARTERIAL BLOOD-PRESSURE |
en |
dc.subject |
PLASMA NOREPINEPHRINE |
en |
dc.subject |
SODIUM RETENTION |
en |
dc.subject |
RECEPTOR-BINDING |
en |
dc.subject |
RABBITS |
en |
dc.subject |
DENERVATION |
en |
dc.subject |
DOGS |
en |
dc.subject |
SENSITIVITY |
en |
dc.title |
What sets the long-term level of renal sympathetic nerve activity - A role for angiotensin II and baroreflexes? |
en |
dc.type |
Journal Article |
en |
dc.identifier.doi |
10.1161/01.RES.0000078346.60663.A0 |
en |
pubs.issue |
12 |
en |
pubs.begin-page |
1330 |
en |
pubs.volume |
92 |
en |
dc.rights.holder |
Copyright: 2003 American Heart Association, Inc |
en |
dc.identifier.pmid |
12764023 |
en |
pubs.end-page |
1336 |
en |
dc.rights.accessrights |
http://purl.org/eprint/accessRights/OpenAccess |
en |
pubs.subtype |
Article |
en |
pubs.elements-id |
6079 |
en |
pubs.org-id |
Bioengineering Institute |
en |
pubs.org-id |
ABI Associates |
en |
pubs.org-id |
Medical and Health Sciences |
en |
pubs.org-id |
Medical Sciences |
en |
pubs.org-id |
Physiology Division |
en |
pubs.record-created-at-source-date |
2010-09-01 |
en |
pubs.dimensions-id |
12764023 |
en |