dc.contributor.author |
Cooling, Michael |
en |
dc.contributor.author |
Hunter, Peter |
en |
dc.contributor.author |
Crampin, Edmund |
en |
dc.coverage.spatial |
Long Beach, CA |
en |
dc.date.accessioned |
2011-11-02T21:39:54Z |
en |
dc.date.issued |
2007-10-01 |
en |
dc.identifier.citation |
The Eight International Conference of Systems Biology. 01 Oct 2007 |
en |
dc.identifier.uri |
http://hdl.handle.net/2292/8577 |
en |
dc.description.abstract |
The transcription factor NFAT acts as a signal integrator for a number of signal transduction pathways in cardiac myocytes that initiates gene expression in the disease Pathological Cardiac Hypertrophy[1]. Here we develop a quantitative mathematical model of the cytoplasmicnuclear-cytoplasmic cycling of NFAT in response to calcium signals in the cardiac myocyte |
en |
dc.relation.ispartof |
The Eight International Conference of Systems Biology |
en |
dc.rights |
Items in ResearchSpace are protected by copyright, with all rights reserved, unless otherwise indicated. Previously published items are made available in accordance with the copyright policy of the publisher. |
en |
dc.rights.uri |
https://researchspace.auckland.ac.nz/docs/uoa-docs/rights.htm |
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dc.title |
Modelling NFAT Cycling Sensitivity in the Cardiac Myocyte |
en |
dc.type |
Conference Poster |
en |
dc.rights.holder |
Copyright: the author |
en |
pubs.author-url |
http://www.icsb07.caltech.edu/proceedings/abstracts/I05.pdf |
en |
dc.rights.accessrights |
http://purl.org/eprint/accessRights/OpenAccess |
en |
pubs.elements-id |
90820 |
en |
pubs.org-id |
Bioengineering Institute |
en |
pubs.org-id |
ABI Associates |
en |
pubs.org-id |
Science |
en |
pubs.org-id |
Science Research |
en |
pubs.org-id |
Maurice Wilkins Centre (2010-2014) |
en |
pubs.record-created-at-source-date |
2010-09-01 |
en |